New Strategies in Stroke Intervention Ionic Transporters, Pumps, and New Channels /
Ischemic brain damage represents a major source of morbidity and mortality in westernized society and poses a significant financial burden on the health care system. To date, few effective therapies have been realized to treat stroke and promising avenues have not proven clinically useful. Recent ev...
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| Format: | Electronic |
| Language: | English |
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Totowa, NJ :
Humana Press,
2010.
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| Series: | Contemporary Neuroscience
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| Online Access: | https://ezaccess.library.uitm.edu.my/login?url=http://dx.doi.org/10.1007/978-1-60761-280-3 |
Table of Contents:
- Basis of ionic dysregulation in cerebral ischemia
- Why have ionotropic and metabotropic glutamate antagonists failed in stroke therapy?
- Mitochondrial channels as potential target for a pharmacological strategy for brain ischemia
- Endoplasmic reticulum calcium homeostasis and neuronal pathophysiology of stroke
- NCX gene product expression and function in brain ischemia
- The role of Na-K-Cl co-transporter in cerebral ischemia
- The Na(+)/H(+) exchanger: a target for therapeutic intervention in cerebral ischemia
- Na+/K+-ATPase as drug target for the treatment of ischemic stroke
- Acid-sensing ion channels (ASICs): new targets for cerebral ischemia
- TRPM7 and TRPC in ischemic CNS injury. - Subtypes of voltage-gated Ca2+ channels and ischemic brain injury
- Role of K+ channels in brain ischemia
- Clinical trials with drugs targeting ionic channels, antiporters, and pumps in ischemic stroke
- Index.