The Ischemic Heart
Over the past three decades, impressive progress in the field of pathogenesis, prevention and therapy of ischemic heart disease has resulted in a marked decline in mortality in the Western World. However, the incidence of this devastating disease is on the rise in developing countries. The Ischemic...
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| Other Authors: | , , , |
| Format: | Electronic |
| Language: | English |
| Published: |
Boston, MA :
Springer US,
1998.
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| Series: | Progress in Experimental Cardiology,
1 |
| Subjects: | |
| Online Access: | View fulltext via EzAccess |
Table of Contents:
- Pathophysiologic Mechanisms of Ischemia-Reperfusion Injury
- Function of Cardiac Myocytes in Ischemicheart Disease: Comparative Changes in Right and Left Ventricular Cells
- Remodeling of Cardiac Myocytes in Chronic Heart Disease
- Hibernating Myocardium
- Length-Dependent Modulation of Cardiac Muscle Contractility in Normoxia, Hypoxia, and Acidosis: Possible Implications for Ischemia
- Roles of Superoxide, Nitric Oxide, and Peroxynitrite in Various Pathological Conditions
- Real-Time Measurement of Nitric Oxide in Coronary Outflow during Transient Myocardial Ischemia and Reperfusion
- Production of Hydrogen Peroxide During Hypoxia-Reoxygenation in Isolated Myocytes
- Oxygen Free Radicals Enhance Ergonovine-Induced Canine Coronary Vasoconstriction
- Structure and Expression of Calponin in Arterial Smooth Muscle Cells
- Status of ?-Adrenoceptors, G-Proteins, and Adenylyl Cyclase in Ischemic Heart Disease
- Oxygen Consumption and Mitochondrial Membrane Potential in Postischemic Myocardium
- Pathophysiological Behavior of the Myocardium in Acute Ischemia and Reperfusion, with Special Emphasis on the Sarcoplasmic Reticulum
- Ca2+ Ion Shifts in Vivo in Reversible and Irreversible Ischemic Injury
- Ionic Mechanisms of Reperfusion Injury
- Mechanism of Inhibition of Na+-H+ Exchanger (NHE1) by ATP Depletion: Implications for Myocardial Ischemia
- The Source and Fate of Protons in the Reperfused Ischemic Heart
- Intracellular Calcium Regulation in Cardiac Cells during Acidosis
- Intracellular pH Regulation and Myocardial Ischemia-Reperfusion Injury
- Regulation of [Na+ i and [Ca2+]i during Myocardial Ischemia and Reperfusion in a Single-Cell Model
- ATP-Sensitive Potassium Channels and Myocardial Ischemia
- Preconditioning and Protection of Ischemia-Reperfusion Injury
- Early and Delayed Protection Against Ventricular Arrhythmias Induced by Preconditioning
- Effects of Ischemic Preconditioning on Na+-Ca2+ Exchanger Activity and Ion Regulation in Isolated Perfused Rat Hearts
- Long-Term but not Short-Term Cardioprotection can be Induced by Preconditioning in Hypercholesterolemia
- Contribution of Endogenous Catecholamines to Preconditioning: Is it through Facilitation of Adenosine Production?
- Hypoxic Preconditioning of Isolated Cardiomyocytes of Adult Rat
- Pharmacological Modification of Ischemic Preconditioning During Percutaneous Transluminal Coronary Angioplasty
- Protection Against Injury Durin Gmyocardial Ischemia and Reperfusion: Concepts and Strategies
- Mechanisms of Cardioprotective Effects of Propranolol Against Reperfusion Injury
- Manganese Superoxide Dismutase Induced by Extracellular Stress Enhances Myocardial Tolerance to Ischemia-Reperfusion
- Thioctic Acid Prevents Oxidativedamage in Isolated Cardiac Myocytes
- Transfection of Heat-Shock Protein 70 Enhances Myocardial Tolerance to Ischemia: Evidence for a Role of HSP70 in Ischemia-Reperfusion Injury
- Calcium but not Amiloride Improves the Mechanical Function of Reperfused One-Week-Old Rabbit Hearts
- Effects of BIBR-277, an Angiotensin-II Type-1 Receptor Antagonist, on Ischemic Myocardial Stunning in Dogs
- Reperfusion 12 Hours after Coronary Occlusion Salvages Myocardium in Dogs: Studies in a Single-Heart Model
- Ecto-5?-Nucleotidase and Cardioprotection
- Limitation of Stunning in Dog Myocardium by the Nucleoside-Nucleotide Mixture OG-VI
- Decrease in Myocardial Ischemic Tolerance with Aging in Fischer 344 Rats
- Underperfusion Injury in Diabetic Rat Hearts: Effects of Norepinephrine and/or Insulin on Stiffness Increase and Abnormal Energy Metabolism
- Do Ketone Bodies Contribute to Protection against Damage Caused by Bothmyocardial Ischemia and Reperfusion Injury?
- Effect of Basic Fibroblast Growth Factor on the Regional Myocardial Blood Flow of Acutely Infarctedmyocardium in Experimental Animals
- Intramyocardial Ejection of Basic Fibroblast Growth Factor Increased Regional Myocardial Blood Flow Andsalvaged Infarcted Myocardium in Dogs
- Effect of Basic Fibroblast Growth Factor on the Regional Myocardial Blood Flow in Acutely Infarcted Myocardium in Rats
- Gene Therapy for Myocardial Infarction
- Adenoviral Vectors as Experimental and Therapeutic Tools
- Prevention of Transplantation-Associated Arteriosclerosis by Antiadhesion Therapy.